Active infective endocarditis
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Active infective endocarditis a clinicopathologic analysis of 137 necropsy patients by Ernest N. Arnett

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Published by Year Book Medical Publishers in Chicago .
Written in English


  • Infective endocarditis.,
  • Pathology.

Book details:

Edition Notes

Includes bibliographical references.

StatementErnest N. Arnett, William C. Roberts.
SeriesCurrent problems in cardiology ;, v. 1, no. 7
ContributionsRoberts, William C. 1932- joint author.
LC ClassificationsRC685.E5 A7
The Physical Object
Pagination76 p. :
Number of Pages76
ID Numbers
Open LibraryOL4599464M
ISBN 100815199155
LC Control Number77362288

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Active infective endocarditis is one of the most serious diseases of the heart. Infection often causes periannular abscess, and may also spread to affect the mitral valve structures. This complication carries a high mortality rate and valve replacement followed by intensive care and antibiotic treatment may be the only option to save patients. Infective endocarditis (IE) is defined as an infection of the endocardial surface of the heart, which may include one or more heart valves, the mural endocardium, or a septal defect. Its intracardiac effects include severe valvular insufficiency, which may lead to intractable congestive heart failure and myocardial abscesses. Introduction. Infective endocarditis (IE) is an infectious and inflammatory process of endothelial lining of the heart structures and valves. It is most commonly caused by bacterial and fungal infections, although non-infective causes of endocarditis occur, this chapter will concentrate on infective causes. patients with active infective endocarditis. Fever is occasion-ally absent in older patients with subacute infective endo-carditis, in patients with terminal renal failure or cerebral haemorrhages, and in patients taking antipyretics or anti-biotics. A new regurgitant murmur should raise the suspicion of infective endocarditis. Complications.

Infective endocarditis (IE) is a life-threatening disease that is associated with high morbidity and mortality. Its long-term prognosis strongly depends on a timely and optimized antibiotic treatment. Certain cases of infective endocarditis are deemed inoperable because of multiorgan failure or extensive cerebral damage from septic emboli, and these patients die of the disease. This study is a retrospective review of a single center experience with surgery for active infective : Fahad Aziz, Sujatha Doddi, Sudheer Penupolu, Simanta Dutta, Anshu Alok. Active infective endocarditis: low mortality associated with early surgical treatment indicate that an early surgical treatment during the active phase of endocarditis where some complication developed and/or it was resistant to antibiotic, may be associated with low mortality and acceptable morbidity. Year Book Medical Publishers Cited by: American Academy of Pediatrics. Prevention of Bacterial Endocarditis. In: Kimberlin DW, Brady MT, Jackson MA, Long SS, eds. Red Book: Report of the Committee on Infectious Diseases. American Academy of Pediatrics; ;

infective endocarditis are significant. It is heterogeneous in etiology, clinical manifestations, and course. Staphylococcus aureus, which has become the predominant causative organism in the developed world, leads to an aggressive form of the disease, often in vulnerable or elderly patient by: Infective endocarditis (IE) has become a ‘surgical disease’ during the last decade since approximately half of all patients are currently operated on during the active phase of the disease. Surgical Treatment of Active Infective Endocarditis, Primary Operation, In-Hospital Mortality – The in-hospital mortality rate for patients who had primary operations for infective endocarditis at Cleveland Clinic are equal to or lower than expected rates, despite a high-risk patient population.   Savage EB, Saha-Chaudhuri P, Asher CR, et al. Outcomes and prosthesis choice for active aortic valve infective endocarditis: analysis of the Society of Thoracic Surgeons Adult Cardiac Surgery : Mahbub Jamil, Ibrahim Sultan, Thomas G. Gleason, Forozan Navid, Michael A. Fallert, Matthew S. Suffo.